Table 1

Over expressed genes as a consequence of chromosome 5p gain in cervical cancer

Gene
Description
Function
Role in cancer
Average fold change#

OSMR
Oncostatin M receptor
Signal transduction
[31]
3.1
BASP1
Brain abundant, membrane attached signal protein 1
DNA binding
-
6.0
C5orf28
Chromosome 5 open reading frame 28
Not known
-
2.9
TRIP13
Thyroid hormone receptor interactor 13
HPV16 E1 protein binding protein
[24]
5.0
NNT
Nicotinamide nucleotide transhydrogenase
Detoxification of ROS
-
3.7
TARS
Threonyl-tRNA synthetase
Aminoacylation of tRNA
-
3.2
PAIP1
Polyadenylate-binding protein-interacting protein 1
Translation activator activity
-
2.3
SDHA
Succinate dehydrogenase complex, subunit A, flavoprotein
Oxidative phosphorylation
-
3.4
NDUFS6
NADH dehydrogenase (ubiquinone) Fe-S protein 6
Oxidative phosphorylation
-
2.3
BRD9
Bromodomain containing 9
Nucleic acid binding
-
2.2
FASTKD3
FAST kinase domains 3
Unknown
-
2.4
RAD1
RAD1 homolog
DNA repair, cell cycle check point
-
3.2
BXDC2
Brix domain containing 2
Ribosomal biosynthesis
-
2.6
SKP2
S-phase kinase-associated protein 2
Mitotic check point
[33]
3.6
C5orf22
Chromosome 5 open reading frame 22
Unknown
-
3.0
POLS
Polymerase (DNA directed) sigma
DNA repair
-
2.8
RNASEN
Ribonuclease III, nuclear
miRNA processing
[27]
2.8

# Fold-change is calculated based on comparison between normal cervical squamous epithelium and tumors with Chr5p gain

Scotto et al. Molecular Cancer 2008 7:58   doi:10.1186/1476-4598-7-58